Aging as an Adaptation
                                                        
                                                          A Proposal

Senescence is a very complicated multi-facet physiological phenomenon that occurs in most tissue over time. Here the senescence of the MHC (major histocompatibility complexes) will be addressed. Contagious diseases can form an universal selective evolutionary force. The focus of this force is on the genes of the MHC. The MHC plays a pleiotropic role, both in the fitness (survival and reproductive success) and in the development of the carrier. The existence of genes for the programming of primary aging is discounted on the basis of evolutionary arguments against the notion that aging has any adaptation value. Adaptation values are not always obvious. Kin selection can result in a very long potential reproductive period for a dominant animal. This extended reproductively would decrease the effective MHC gene pool size. Eventually such specie will become extinct with the onslaught of new infectious challenges. Animals capable of aging will become the dominant life form because of a more vigorous immune system. One can conclude that aging is the indirect consequence of bacterial and virul pressure.

J. Coetzee PhD

 For example the Embodied capital and grandmother effect. Phenoptosis and progeria in salmon is well known.
 The hypothesis in this document will be  based on existing published peer-reviewed research. It had a limited review.